The KCNQ1 voltage-gated potassium (Kv) channel pore-forming (α) subunit is ubiquitously expressed and linked to life-threatening human disorders including Long QT syndrome, atrial fibrillation and diabetes. KCNQ1 exhibits a high degree of functional flexibility enabled by co-assembly with KCNE family β subunits, facilitating roles both in excitable cell repolarization, and as a constitutively active K+…
Kv channel-solute transporter interactions
